Unlocking thalidomide response as a hemoglobin F augmentation agent in transfusion-dependent β-thalassemia: the genetic impact of BCL11A, HBS1L-MYB, and XmnI polymorphism

Diagnostic Pathology · Published 2026-07-07 · DOI 10.1186/s13000-026-01797-5

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Authors (4)

Iram Nazir, Muhammad Younus Jamal Siddiqi, Maeesa Wadood, Muhammad Rizwan

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Year

2026

Citation

Nazir, I., Siddiqi, M., Wadood, M., et al. (2026). Unlocking thalidomide response as a hemoglobin F augmentation agent in transfusion-dependent β-thalassemia: the genetic impact of BCL11A, HBS1L-MYB, and XmnI polymorphism. Diagnostic Pathology. https://doi.org/10.1186/s13000-026-01797-5

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