A critical residue mediates proper assembly and gating of GIRK2 channels

Journal of General Physiology · Published 2025-11-22 · DOI 10.1085/jgp.202513817

Authors being retrieved — see the publisher record. https://doi.org/10.1085/jgp.202513817

Abstract

PubPorta does not have an abstract for this article yet.

Read the article at the publisher →

Publication details

Year: 2025

Related articles

Cholesterol pools cooperate to modulate HCN channels · Journal of General Physiology · 2026 · Same journal
Dysferlin’s C2A domain supports normal Ca2+ signaling and membrane repair in dysferlin-null myofibers · Journal of General Physiology · 2026 · Same journal
On the mechanism of K+ transport through the inter-subunit tunnel of KdpFABC · Journal of General Physiology · 2025 · Same journal
Combining elastic network models and linear response theory as tool to understand the global dynamics in allosteric regulation of HCN channels · Journal of General Physiology · 2026 · Same journal
Coincidence detection supported by electrical synapses is shaped by the D-type K+ current · Journal of General Physiology · 2026 · Same journal
Evolution, hibernation, and inactivation of voltage-gated Na channels · Journal of General Physiology · 2026 · Same journal
Model of ocular surface ion and water transport predicts efficacy of dry eye therapeutics · Journal of General Physiology · 2026 · Same journal
Inhibition of CaV1.4 channels by CaV3 channel antagonists ML218 and Z944 · Journal of General Physiology · 2025 · Same journal
Optical control of PI(4,5)P2 sensitivity of ion channels by manipulation of single lysine residue · Journal of General Physiology · 2025 · Same journal
On the mechanism of hypomagnesemia with treatment-resistant seizures caused by variants of the Na+,K+-ATPase α1 subunit (ATP1A1) · Journal of General Physiology · 2026 · Same journal